Chair of Biochemistry and Molecular Biology

    Publications

    E.K. Herter, M. Stauch, M. Gallant, E. Wolf, T. Raabe and P. Gallant (2015). snoRNAs are a novel class of biologically relevant Myc targets. BMC Biology (in press)

    A. Hovhanyan, E. Herter, J. Pfannstiel, P. Gallant and T. Raabe (2014). Drosophila Mbm is a nucleolar Myc and CK2 target required for ribosome biogenesis and cell growth of central brain neuroblasts. Mol. Cell. Biol. 34, 1878.

    P. Gallant (2013). Myc Function in Drosophila. Cold Spring Harb Perspect Med, a014324.

    P. Bellosta & P. Gallant (2010). Myc Function in Drosophila. Genes Cancer 1, 542.

    M. Furrer, M. Balbi, M. Albarca-Aguilera, M. Gallant, W. Herr, & P. Gallant (2010). Drosophila Myc interacts with Host Cell Factor (dHCF) to activate transcription and control growth. J. Biol. Chem. 285, 39623-39636

    P. Gallant & D. Steiger (2009). Extra view: Myc’s secret life without Max. Cell Cycle 8 (23), 3848-3853.

    P. Gallant (2009). Drosophila Myc. Advances in Cancer Research 103, 111-144.

    D. Schwinkendorf & P. Gallant (2009). The conserved Myc box 2 and Myc box 3 regions are important, but not essential, for Myc function in vivo. Gene 436, 90-100.

    D. Steiger, M. Furrer, D. Schwinkendorf & P. Gallant (2008). Max-independent functions of Myc in Drosophila melanogaster. Nature Genetics 40, 1084-1091.

    L. Montero, N. Müller and P. Gallant (2008). Induction of apoptosis by Drosophila Myc. Genesis 46, 104-111.

    H. Stocker and P. Gallant (2008). Getting started: An overview on raising and using Drosophila. In “Drosophila: Methods and Protocols” (C. Dahmann, editor). Humana Press, Totowa, NJ (USA).

    P.Gallant (2007). Control of transcription by Pontin and Reptin. Trends Cell Biol. 17, 187-192.

    P.Gallant (2006). Myc / Max / Mad in invertebrates - the evolution of the Max network. Curr. Top. Micro. Imm. 320: 237-254.

    C. Benassayag, L. Montero, N. Colombie, P. Gallant, D. Cribbs, D. Morello (2005). Human c-Myc isoforms rescue a lethal Drosophila dmyc mutation and differentially regulate transcription, growth, proliferation and apoptosis in vivo. Mol. Cell. Biol. 25, 9897.

    P. Bellosta, T. Hulf, S.B. Diop, F. Usseglio, J. Pradel, D. Aragnol, and P. Gallant (2005). Myc interacts genetically with Tip49/Pontin to control growth and proliferation during Drosophila development. Proc. Natl. Acad. Sci. USA 102, 11799-11804.

    T. Hulf, P. Bellosta, M. Furrer, D. Steiger, D. Svensson, A. Barbour, P. Gallant (2005). Whole-genome analysis reveals a strong positional bias of conserved dMyc-dependent E-boxes. Mol. Cell. Biol. 25, 3401.

    P.Gallant (2005). Myc, cell competition and compensatory proliferation. Cancer Research 65, 6485-6487.

    C. De La Cova, M. Abril, P. Bellosta, P. Gallant, and L.A. Johnston (2004). Drosophila myc regulates organ size by inducing cell competition. Cell 117: 107-16.

    L. A. Johnston and P.Gallant (2002). Control of growth and organ size in Drosophila. BioEssays 24, 54-64.

    L.A. Johnston, D.A. Prober, B.A. Edgar, R.N. Eisenman and P. Gallant (1999). Drosophila myc regulates cellular growth during development. Cell 98, 779-790.

    S. Zaffran, A. Chartier, P. Gallant, M. Astier, N. Arquier, D. Doherty, D. Gratecos, and M. Semeriva, M. (1998): A Drosophila RNA helicase gene, pitchoune, is required for cell growth and proliferation and is a potential target of d-Myc. Development 125, 3571-3584.nal repression to cell differentiation. Cold Spring Harbor Symp. Quant. Biol., 63: 423-433.

    P.Gallant, Y. Shiio, P.F. Cheng, S.M. Parkhurst, and R.N. Eisenman (1996). Myc and Max homologs in Drosophila melanogaster. Science 274: 1523-1527.

    Kontakt

    Lehrstuhl für Biochemie und Molekularbiologie
    Am Hubland
    97074 Würzburg

    Tel.: +49 931 31-84112
    E-Mail

    Suche Ansprechpartner

    Hubland Süd, Geb. B1 Hubland Nord, Geb. 32 Julius-von-Sachs-Platz 2 Fabrikschleichach Hubland Süd, Geb. B2 Campus Medizin